review Open AccessTop 1% cited
EMT: 2016
Cell · 2016 · Vol. 166(1) · pp. 21–45
M. Ángela Nieto✉(Instituto de Neurociencias)Ruby Yun‐Ju Huang(National University of Singapore)Rebecca Jackson(National University of Singapore)Jean Paul Thiery(National University Cancer Institute, Singapore)
Cancer Cells and MetastasisFOXO transcription factor regulationMicroRNA in disease regulationBiologyEpithelial–mesenchymal transitionParallelsTumor progressionEmbryonic stem cellPhenotypeMesenchymal stem cellComputational biologyMetastasisNeuroscience
MeSH terms
AnimalsFibrosisHumansNeoplasmsTranscription, GeneticEpigenesis, GeneticEmbryonic DevelopmentEpithelial-Mesenchymal Transition
Citations
4,367
FWCI
183.13
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References
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References
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Nature Medicine · 2015 · 3,213 citations
Pancreatic cancer exosomes initiate pre-metastatic niche formation in the liver
Nature Cell Biology · 2015 · 2,668 citations
Quantitative expression of Oct-3/4 defines differentiation, dedifferentiation or self-renewal of ES cells
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Implications of the Hybrid Epithelial/Mesenchymal Phenotype in Metastasis
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Molecular mechanisms of epithelial–mesenchymal transition
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An EMT spectrum defines an anoikis-resistant and spheroidogenic intermediate mesenchymal state that is sensitive to e-cadherin restoration by a src-kinase inhibitor, saracatinib (AZD0530)
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