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PPARγ Is Required for Placental, Cardiac, and Adipose Tissue Development
Molecular Cell · 1999 · Vol. 4(4) · pp. 585–595
Yaacov Barak(Salk Institute for Biological Studies)Michael C. Nelson(Salk Institute for Biological Studies)Estelita S. Ong(Salk Institute for Biological Studies)Ying Jones(University of California San Diego)Pilar Ruiz‐Lozano(University of California San Diego)Kenneth R. Chien(University of California San Diego)Alan KoderRonald M. Evans✉(Salk Institute for Biological Studies)
Peroxisome Proliferator-Activated ReceptorsPancreatic function and diabetesRNA modifications and cancerBiologyAdipogenesisTrophoblastPlacentaAdipose tissuePeroxisome proliferator-activated receptorHeart developmentEndocrinologyReceptorInternal medicine
MeSH terms
Adipose TissueAnimalsCell DifferentiationFetal ViabilityHeartLipodystrophyLiverMyocardiumNuclear ProteinsPlacentaPlacentationPloidiesRNA, MessengerTranscription FactorsIn Situ Hybridization
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References
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An Antidiabetic Thiazolidinedione Is a High Affinity Ligand for Peroxisome Proliferator-activated Receptor γ (PPARγ)
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