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Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion

Nature Cell Biology · 2009 · Vol. 11(8) · pp. 973–979

Françis Rodier✉(Lawrence Berkeley National Laboratory)Jean‐Philippe Coppé(Lawrence Berkeley National Laboratory)Christopher K. Patil(Lawrence Berkeley National Laboratory)Wieteke A. M. Hoeijmakers(Radboud University Medical Center)Denise P. Muñoz(Buck Institute for Research on Aging)Saba R. Raza(Lawrence Berkeley National Laboratory)Adam Freund(University of California, Berkeley)Eric Campeau(University of Massachusetts Chan Medical School)Albert R. Davalos(Lawrence Berkeley National Laboratory)Judith Campisi(Buck Institute for Research on Aging)

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Telomeres, Telomerase, and Senescence Retinoids in leukemia and cellular processes NF-κB Signaling PathwaysDNA damageSenescenceSecretionCell biologyBiologyCytokineCell cycleCell cycle checkpointCancer cellCell

MeSH terms

Cell LineCells, CulturedDNA DamageDNA-Binding ProteinsEnzyme-Linked Immunosorbent AssayFibroblastsHumansMaleMicroscopy, FluorescenceNuclear ProteinsTransfectionBlotting, WesternSignal TransductionInterleukin-6Tumor Suppressor Protein p53

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